Frank Cucinotta (Health Physics and Diagnostic Sciences) has had his work published in several journals. He is the senior author of "Analysis of the Lymphocytopoiesis Dynamics in Nonirradiated and Irradiated Humans: A Modeling Approach." The article describes how an existing mathematical model of the lymphocytopoiesis system was extended to predict the dynamics of this system within people who have and have not been exposed to radiation. The model was also shown to be capable of predicting the system's dynamics in those with acute and chronic radiation exposure over a wide range of doses and dose rates. The article appeared in the March issue of the journal Radiation Research. He also is the senior author of "Investigation of Switch from ATM to ATR Signaling at the Sites of DNA Damage Induced by Low and High LET Radiation," which provides new knowledge about how ATM and ATR regulate the cellular DNA damage response and differences in this response due to the types of radiation, such as gamma-rays or heavy ions, used for cancer therapy or comprising cosmic rays. The article appeared in the December issue of the journal DNA Repair. He also co-authored "OCT4 as a Target of miR-34a Stimulates p63 but Inhibits p53 to Promote Human Cell Transformation," which discusses the molecular pathways that control radiation neoplastic transformation, or cancer formation. The article appeared in the Jan. 23 issue of Cell Death & Disease. He is also the senior author of "Modeling Damage Complexity-Dependent Non-Homologous End-Joining Repair Pathway," which appeared in the February 10, issue of PLOS ONE. The article focuses on using non-homologous end-joining (NHEJ) to repair double strand breaks in cellular DNA that was caused by ionizing radiation. Since the NHEJ repair pathway involves several repair proteins, the study examined how the choice of proteins is determined by the complexity of the double strand break.